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Annals of Botany 2008 102(3):vii; doi:10.1093/aob/mcn148
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© The Author 2008. Published by Oxford University Press on behalf of the Annals of Botany Company. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

John Bryant takes a closer look at some of this month's Original Articles

J. A. Bryant, Professor

University of Exeter, UK
E-mail j.a.bryant@exeter.ac.uk

The first 10% of the full text of this article appears below.

Gliadin gene control – comfort comes closer for celiac sufferers


Figure 1
There is little doubt that the incidence of diagnosis of celiac (cœliac) disease – mainly caused by allergy to wheat {alpha}-gliadins – is increasing. Whether this increase arises, as some suggest, from the way that bread is manufactured on a large scale or whether detection of the condition has improved is not clear. Van Herpen et al. (a joint UK–Netherlands research team; pp. 331–342) cite previous results showing that the allergic reactions exhibited by sufferers are elicited by particular epitopes in the {alpha}-gliadins encoded in the D-genome and, to a lesser extent, in the A-genome of wheat. Gliadins encoded by the B-genome are much less allergenic. Further, there is evidence that the timings of deposition during grain development of the {alpha}-gliadins encoded by the three genomes are . . . [Full Text of this Article]

Sugar maple succumbs to strength-sapping stress

Can CAM cope with copious carbon dioxide?

Light and leaves – periwinkle proves DAT's the way to do it


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